I am creating a thread to promote awareness about how hormones in drugs, pesticides/herbicides, etc are wreaking havoc on people's brains and bodies.

"They" don't warn people that the procedures, drugs and the poisoned water could damage them in ways they could never imagine.

The Increasing Prevalence in Intersex Variation from Toxicological Dysregulation in Fetal Reproductive Tissue Differentiation and Development by Endocrine-Disrupting Chemicals
[www.ncbi.nlm.nih.gov]

Tai: Yet one more urgent reason to distill your own spring water.

Tai: I don't think it's a coincidence that the Baphomet (satanic hemaphrodite/part beast-part human figure) image has been emerging on government properties and in hollywood, as Monsanto has been trying to control the world's seed supply and making plants dependent on pesticides. SEED/SEMEN/EGGS.

Intersex variation is not bad in itself, as this happens in nature naturally, eg Hyenas. But when it's forced on a growing baby that would otherwise be regular, that's sheer evil.

Maternal progestin intake and risk of hypospadias.
[www.ncbi.nlm.nih.gov]

RESULTS: Forty-two case mothers (8.4%) and 31 control mothers (2.4%) reported any pregnancy-related progestin intake from 4 weeks before through 14 weeks after conception,
This study found that pregnancy-related intake of progestins was associated with increased hypospadias risk.

Tai: with severe hypospadias, a boy could be subject to repeated surgeries and have his quality of life severely diminished.

[www.cdc.gov]
Facts about Hypospadias

Progestins 101
What you should know about the synthetic form of progesterone.
[helloclue.com]

[www.ncbi.nlm.nih.gov]
In vitro fertilization is associated with an increased risk of hypospadias.

[rscbayarea.com]
Are Birth Defects More Common with IVF? YES

Tai: where is the future of conception heading?

Genetic testing’s role in reducing birth defects
The "good news" [sic] is that we can often address genetic problems using preimplantation genetic screening (PGS). This can be done with IVF to detect many chromosomal abnormalities. Another genetic screening method, preimplantation genetic diagnosis (PGD) can search for a specific genetic disease, such as muscular dystrophy, if we know the parent(s) are at risk of carrying that disease.
Tai: small print: price tag 8k in addition to the 20k for IVF

With PGS and PGD, we can identify genetically abnormal IVF embryos that would result in birth defects, or failed pregnancy, and not implant those in the mother. After a successful pregnancy in which genetic testing was not used, other types of screening during early pregnancy, such as amniocentesis, can detect the majority of the small percent of IVF babies who do have an abnormality.

Tai: abortion goes hand in hand with IVF

[www.webmd.com]
IVF Babies and Major Birth Defects

Tai: the thing is, these children would not have been born because the couple was infertile. So even though it's not that much higher than average, it's still more than nature would have allowed. Natural healing can increase fertility. A couple should spend a year and detox and tonify their bodies and get pregnant naturally. There are rare cases where anatomy will not allow the natural; these could be argued for IVF, but where is IVF taking humans in the long run?

[www.independent.co.uk]
Children born through IVF face higher risk of cardiovascular disease, warns study

“There is growing evidence that artificial reproduction techniques (ART) alters the blood vessels in children, but the long-term consequences were not known,” said Dr Emrush Rexhaj, a blood pressure expert and lead author of the study.

“We now know that this places children [born through artificial reproduction] at a six times higher rate of hypertension (high blood pressure) than children conceived naturally.”

Dr Rexhaj told The Independent: “This is the first demonstration of increased prevalence of a cardiovascular disease [in children conceived through IVF].”

But he said a 2014 study has suggested these patients may also be more at risk of Type 2 diabetes, adding: “There is already evidence showing insulin resistance in this population.”

There are an estimated six million people alive who were conceived with artificial reproduction techniques worldwide and in July, Louise Brown, the first child born through the IVF, celebrated her 40th birthday.

He added: “It may be that the first few days of exposure of an embryo to artificial culture media may affect a number of developing organs, including the heart and blood vessels.”



Edited 3 time(s). Last edit at 11/11/2019 05:19PM by Tai.

The crazy thing about this drug is it can create ambiguous genitalia in babies whose mothers were exposed to this drug, even handling it.

[www.accessdata.fda.gov]

CONTRAINDICATIONS PROPECIA is contraindicated in the following: Pregnancy. Finasteride use is contraindicated in women when they are or may potentially be pregnant. Because of the ability of Type II 5?-reductase inhibitors to inhibit the conversion of testosterone to DHT, finasteride may cause abnormalities of the external genitalia of a male fetus of a pregnant woman who receives finasteride. If this drug is used during pregnancy, or if pregnancy occurs while taking this drug, the pregnant woman should be apprised of the potential hazard to the male fetus. (See also WARNINGS, EXPOSURE OF WOMEN - RISK TO MALE FETUS; and PRECAUTIONS, Information for Patients and Pregnancy.) In female rats, low doses of finasteride administered during pregnancy have produced abnormalities of the external genitalia in male offspring.

EXPOSURE OF WOMEN - RISK TO MALE FETUS Women should not handle crushed or broken PROPECIA tablets when they are pregnant or may potentially be pregnant because of the possibility of absorption of finasteride and the subsequent potential risk to a male fetus. PROPECIA tablets are coated and will prevent contact with the active ingredient during normal handling, provided that the tablets have not been broken or crushed.

[www.dailymail.co.uk]

Woman, 31, with severe allergy to penicillin goes into anaphylactic shock after swallowing her partner's semen 'because was on antibiotics'

Tai: if women are pregnant or going to get pregnant, they need to know how serious exposure is to this anti-baldness drug and how they can get exposed to it, even through semen perhaps.

[www.omicsonline.org]

Tai: 19 exposed pregnanies and 6 abortions/miscarriages = 13 healthy ones out of how many thousands?

[www.vice.com]

I Need to Quit Hair Loss Drugs Before They Kill Me

[elemental.medium.com]

My Life Has Been Ruined by an Anti-Baldness Drug
I’ve endured depression, anxiety, headaches, memory loss, insomnia, blurred vision, and impotence since I was 19

[www.dailymail.co.uk]

[www.thesun.co.uk]
FOLLICLE DYSFUNCTION Drug that banishes baldness ruins men’s love lives – leaving them IMPOTENT

[sperlingprostatecenter.com]



Edited 4 time(s). Last edit at 11/11/2019 05:58PM by Tai.

This is the history of Finasteride

And by the way, I read that in some primitive tribes, this is known to occur where the male genitalia only emerges at puberty. So this shows how wrong it is to perform surgery on a xy baby with no penis

[www.bbc.com]

The discovery of a small community in the Dominican Republic, where some males are born looking like girls and only grow penises at puberty, has led to the development of a blockbuster drug that has "helped" [sic] millions of people, writes Michael Mosley.

Tai: there you have it: MILLIONS of men have taken Finasteride

When Imperato-McGinley investigated the Guevedoces she discovered the reason they don't have male genitalia when they are born is because they are deficient in an enzyme called 5-alpha-reductase, which normally converts testosterone into dihydro-testosterone.

This deficiency seems to be a genetic condition, quite common in this part of the Dominican Republic, but vanishingly rare elsewhere. So the boys, despite having an XY chromosome, appear female when they are born. At puberty, like other boys, they get a second surge of testosterone. This time the body does respond and they sprout muscles, testes and a penis.

Imperato-McGinley's thorough medical investigations showed that in most cases their new, male equipment seems to work fine and that most Guevedoces live out their lives as men, though some go through an operation and remain female.

Another thing that Imperato-McGinley discovered, which would have profound implications for many men around the world, was that the Guevedoces tend to have small prostates.

This observation, made in 1974, was picked up by Roy Vagelos, head of research at the multinational pharmaceutical giant, Merck. He thought this was extremely interesting and set in progress research which led to the development of what has become a best-selling drug, finasteride, which blocks the action of 5-alpha-reductase, mimicking the lack of dihydro-testosterone seen in the Guevedoces.

My wife, who is a GP, routinely prescribes finasteride as it is an effective way to treat benign enlargement of the prostate, a real curse for many men as they get older. Finasteride is also used to treat male pattern baldness.

Tai: Give me a break, stinging nettle root, acupuncture, low fat plant diet, flax seeds (among so many other natural herbs) are the TRUE effective ways for BPH, not a drug that is going to make a man impotent, and might cause his unborn baby to have ambiguous genitals, not to mention the many men that committed suicide from it. What about the oath, Do No Harm?

Back to article:
A final interesting observation that Imperato-McGinley made was that these boys, despite being brought up as girls, almost all showed strong heterosexual preferences. She concluded in her seminal paper that hormones in the womb matter more than rearing when it comes to your sexual orientation.



Edited 3 time(s). Last edit at 11/11/2019 06:24PM by Tai.

Hormone Disruptors Linked To Genital Changes and Sexual Preference

[www.loe.org]

Scientists are continuing to sound the alarm about some common chemicals, including the herbicide atrazine, and link them to changes in reproductive health and development. Endocrine disrupting toxic chemicals have been found to feminize male frogs and cause homosexual behavior. Ashley Ahearn reports on how these substances may be affecting human development and behavior.

[news.berkeley.edu]

Pesticide atrazine can turn male frogs into females

Atrazine, one of the world’s most widely used pesticides, wreaks havoc with the sex lives of adult male frogs, emasculating three-quarters of them and turning one in 10 into females, according to a new study by University of California, Berkeley, biologists.

The 75 percent that are chemically castrated are essentially “dead” because of their inability to reproduce in the wild, reports UC Berkeley’s Tyrone B. Hayes, professor of integrative biology.

“These male frogs are missing testosterone and all the things that testosterone controls, including sperm. So their fertility is as low as 10 percent in some cases, and that is only if we isolate those animals and pair them with females,” he said. “In an environment where they are competing with unexposed animals, they have zero chance of reproducing.”

The 10 percent or more that turn from males into females – something not known to occur under natural conditions in amphibians – can successfully mate with male frogs but, because these females are genetically male, all their offspring are male.

“When we grow these guys up, depending on the family, we will get anywhere from 10 to 50 percent females,” Hayes said. “In a population, the genetically male females can decrease or wipe out a population just because they skew sex ratios so badly.”

Though the experiments were performed on a common laboratory frog, the African clawed frog (Xenopus laevis), field studies indicate that atrazine, a potent endocrine disruptor, similarly affects frogs in the wild, and could possibly be one of the causes of amphibian declines around the globe, Hayes said.

Hayes and his UC Berkeley colleagues report their results in this week’s online early edition of the journal Proceedings of the National Academy of Sciences. In last week’s issue of the Journal of Experimental Biology, Hayes and colleagues published a review of the possible causes of a worldwide decline in amphibian populations, concluding that atrazine and other hormone-disrupting pollutants are a likely contributor because they affect recruitment of new individuals and make amphibians more susceptible to disease.

“These kinds of problems, like sex-reversing animals skewing sex ratios, are much more dangerous than any chemical that would kill off a population of frogs,” he said. “In exposed populations, it looks like there are frogs breeding but, in fact, the population is being very slowly degraded by the introduction of these altered animals.”

Some 80 million pounds of the herbicide atrazine are applied annually in the United States on corn and sorghum to control weeds and increase crop yield, but such widespread use also makes atrazine the most common pesticide contaminant of ground and surface water, according to various studies.

More and more research, however, is showing that atrazine interferes with endocrine hormones, such as estrogen and testosterone – in fish, amphibians, birds, reptiles, laboratory rodents and even human cell lines at levels of parts per billion. Recent studies also found a possible link between human birth defects and low birth weight and atrazine exposure in the womb.

As a result of these studies, the Environmental Protection Agency (EPA) is reviewing its regulations on use of the pesticide. Several states are considering banning atrazine, and six class action lawsuits have been filed seeking to eliminate its use. The European Union already bars the use of atrazine.

Hayes’s studies in the early 2000s were the first to show that the hormonal effects of atrazine disrupt sexual development in amphibians. Working with the African clawed frog, Hayes and his colleagues showed in 2002 that tadpoles raised in atrazine-contaminated water become hermaphrodites – they develop both female (ovaries) and male (testes) gonads. This occurred at atrazine levels as low as 0.1 parts per billion (ppb), 30 times lower than levels allowed in drinking water by the EPA (3 ppb).

Subsequent studies showed that native leopard frogs (Rana pipiens) collected from atrazine-contaminated streams in the Midwest, including from areas up to 1,000 miles from where atrazine is applied, often had eggs in their testes. And many males had lower testosterone levels than normal females and smaller than normal voice boxes, presumably limiting their ability to call mates.

Hayes’ research also established that many frogs in Midwestern streams contaminated by atrazine and other pesticides have compromised immune systems, leading to increased mortality from bacterial disease.

Those early studies were hampered by the inability to easily distinguish genetically male from genetically female frogs. Male frogs have two identical sex chromosomes (ZZ) while females have both a Z and a W – the opposite of XX female and XY male humans. But because all frog chromosomes look the same under a light microscope, it’s not simple to distinguish male from female.

To overcome this, Hayes’ colleague Roger Liu developed a line of all-male frogs so that the genetics would be unequivocal.

“Before, we knew we got fewer males than we should have, and we got hermaphrodites. Now, we have clearly shown that many of these animals are sex-reversed males,” Hayes said. “We have animals that are females, in the sense that they behave like females: They have estrogen, lay eggs, they mate with other males. Atrazine has caused a hormonal imbalance that has made them develop into the wrong sex, in terms of their genetic constitution.”

Coincidentally, another lab in 2008 discovered a sex-linked genetic marker in Xenopus, which has allowed Hayes to confirm the genetic sex of his frogs.

In Hayes’ study, where 40 frogs lived for about three years after hatching in water with 2.5 ppb atrazine, about 10 percent of the frogs appeared to be resistant to the effects of the pesticide. In ongoing studies, Hayes is investigating whether this apparent resistance is inherited, as well as whether the sex-reversed males have more susceptible offspring.

Syngenta, which manufactures atrazine, disputes many of these studies, including Hayes’, that show adverse effects of the pesticide. But Hayes said that “when you have studies all over the world showing problems with atrazine in every vertebrate that has been looked at – fish, frogs, reptiles, birds, mammals – all of them can’t be wrong.”

“What people have to realize is that, just as with taking pharmaceuticals, they have to decide whether the benefits outweigh the costs,” he said. “Not every frog or every human will be affected by atrazine, but do you want to take a chance, what with all the other things that we know atrazine does, not just to humans but to rodents and frogs and fish?”

Hayes’ long-term studies of the effects of atrazine on frogs have been assisted by many UC Berkeley students, including co-authors on the current paper: undergraduates Vicky Khoury, Anne Narayan, Mariam Nazir, Andrew Park, Lillian Adame and Elton Chan; and graduate students Travis Brown, Daniel Buchholz, Sherrie Gallipeau and Theresa Stueve.

The work was funded by the Park Water Co., Mitch Kapor, Freada Klein, the Mitch Kapor Foundation, the David Foundation, the Cornell-Douglas Foundation, the Wallace Foundation, the UC Berkeley Class of ’43 endowed chair and the Howard Hughes Biology Fellows Program.



Edited 1 time(s). Last edit at 11/11/2019 06:31PM by Tai.

[diethylstilbestrol.co.uk]

Posted on 31/08/2011 by DES Daughter
Gender Identity and DES Exposure

Several published studies in the medical literature on psycho-neuro-endocrinology have examined the hypothesis that prenatal exposure to estrogens (including Diethylstilbestrol) may cause significant developmental impact on sexual differentiation of the brain and on subsequent behavioural and gender identity development in exposed males and females. There is significant evidence linking prenatal hormonal influences on gender identity and transsexual development

In 1999, Dr. Scott Kerlin (founder of the DES Sons International Network) began researching the effects of Di-Ethyl Stilbestrol® on the health of genetic males who had been exposed prenatally. A substantial amount of research had been done on women who had been exposed but relatively little had been done on men and DES sons. When it became apparent that a significant portion of his research group were either transsexual, transgendered or intersexed, he began to explore the possibility of a connection between prenatal DES exposure and gender variance. Dr. Kerlin is not the first researcher to note a correlation between DES exposure and feminized behaviour in genetic males; studies go back as far as 1973. However, Dr. Kerlin has delved much deeper than those who came before.

Radio Interview: DES Exposure and Gender Variance
[diethylstilbestrol.co.uk]
Dr. Dana Beyer is the medical advisor and web manager of the DES Sons International Network, on the effects of endocrine disrupting compounds such as Diethylstilbestrol, DDT, phthalates and bisphenol A, on human sexuality and reproduction, as well as providing personal support and mentoring. In 2005 she presented a breakthrough paper, with her colleagues Dr. Scott Kerlin and Dr. Milton Diamond, to the International Behavioural Development Symposium, delineating the impact Di-Ethyl Stilbestrol® has had in causing intersex and gender variations in human beings.


Sky Shine (@BrightBlueCielo)
28/03/2017 at 00:17

My mother took DES. I was born in 1971. I’m transgender and born with PAIS.


[diethylstilbestrol.co.uk]

Prenatal exposure to DES linked to intersexed development in males

Did Diethylstilbestrol cause intersexed development in the DES Sons by blocking testicular testosterone production ?

[diethylstilbestrol.co.uk]

Diethylstilbestrol DES and Gender Identity Studies

Source studies of gender identity, feminization, transsexualism, and sexual orientation in the DES-exposed.

2016 video Did the DES drug make some children transgender ?
[diethylstilbestrol.co.uk]
“And it was one of those moments… when you just go, ‘My God, it really is so obvious’.”
2016 video Gender assignment : something in the mind, as opposed to the body.
“Until there is a funded a DES transgender link study, there won’t be a definitive answer.”
2016 video Real Talk with a DES Son.
“As a male, I was prenatally exposed to medically prescribed hormones.”
2016 video The Controversy about DES and Gender Identity.
“A very good possibility that DES is what caused me to be trans.”
2016 video The possible link between DES and being transgender later in life.
“This is all about education. The more people know, the better off everyone is.”
2016 video Thousands of people believe DES is the reason they are transgender.
“We could not talk about this because it was too scary.”
2015 Early exposure of 17?-ethynylestradiol and diethylstilbestrol induces morphological changes and alters ovarian steroidogenic pathway enzyme gene expression in catfish, Clarias gariepinus.
2015 In utero exposure to the oestrogen mimic diethylstilbestrol disrupts gonadal development in a viviparous reptile.
2014 Fetal programming of sexual development and reproductive function.
2014 Our stolen figures: The interface of sexual differentiation, endocrine disruptors, maternal programming, and energy balance.
2013 Prenatal exposure to DES linked to intersexed development in males.
2012 Estrogen receptor-? mediates diethylstilbestrol-induced feminization of the seminal vesicle in male mice.
2012 Short-term study investigating the estrogenic potency of diethylstilbesterol in the fathead minnow.
2012 Specific morphogenetic events in mouse external genitalia sex differentiation are responsive/dependent upon androgens and/or estrogens.
2011 Gender Identity and DES Exposure.
2011 book : Prenatal exposure to progesterone suppresses reproduction in male mice.
2011 Sexual differentiation of human behavior: Effects of prenatal and pubertal organizational hormones.
2011 Sexual differentiation of the human brain: relation to gender identity, sexual orientation and neuropsychiatric disorders.
2010 Gonadal hormones and sexual differentiation of human behavior : Effects on psychosexual and cognitive development.
2010 Sexual differentiation of the human brain in relation to gender identity and sexual orientation.
2009 Animal model for age- and sex-related genotoxicity of diethylstilbestrol.
2009 Clinical implications of the organizational and activational effects of hormones.
2009 book : The Riddle of Gender – Science, Activism, and Transgender Rights.
2008 DES’ other Daughters : Neglected Evidence of Prenatal Gender Development.
2008 Effects of Gestational Diethylstilbestrol Treatment on Male and Female Gonads during Early Embryonic Development.
2008 Environmental endocrine disrupters.
2007 Secondary sex ratio among women exposed to diethylstilbestrol in utero.
2007 Time to pregnancy and secondary sex ratio in men exposed prenatally to diethylstilbestrol.
2006 Atypical Gender Development – A Review.
2006 audio : Dr. Scott Kerlin talks about Diethylstilbestrol DES.
2005 Are EDCs Blurring Issues of Gender?
2005 audio : Dr. Dana Beyer talks about Diethylstilbestrol DES.
2005 Effects of maternal exposure to a low dose of diethylstilbestrol on sexual dimorphic nucleus volume and male reproductive system in rat offspring.
2005 Prenatal DiEthylStilbestrol Exposure in Males and Gender-related Disorders – Results from a 5-year study.
2004 Basic Statistics and Findings on DES Sons Participating in the DES Sons International Network Between 1999 and 2004.
2004 Children’s search for gender cues : Cognitive perspectives on gender development.
2004 DES Sons and Gender Dysphoria, Transsexualism, Disorders of Sexual Differentiation.
[diethylstilbestrol.co.uk]
2004 DES Sons and the Significance of Gender Identity: Statistics of Individuals with Gender-related Issues or Outcomes Among Network Members.
2004 Discordant sexual identity in some genetic males with cloacal exstrophy assigned to female sex at birth.
2004 Disorders of sex differentiation caused by exogenous hormones.
2004 Gender Benders and Endocrine Disruptors around You.
2004 Prevalence of Transsexualism, Transgenderism, Gender Dysphoria, or Intersex among DES-Exposed.
2004 Sexual differentiation of the human brain : relevance for gender identity, transsexualism and sexual orientation.
2004 The Presence of Gender Dysphoria, Transsexualism, and Disorders of Sex Differentiation in Males Prenatally Exposed to Diethylstilbestrol.
2004 Transsexualism : An Unacknowledged Endpoint of Developmental Endocrine Disruption?
2003 Diethylstilbestrol exposure and feminization in males.
2003 Effects on gender identity of prenatal androgens and genital appearance : evidence from girls with congenital adrenal hyperplasia.
2003 Psychosexual characteristics of men and women exposed prenatally to diethylstilbestrol.
2002 Cognitive theories of early gender development.
2002 Environmental endocrine disrupters and disorders of sexual differentiation.
2002 Estrogens and environmental estrogens.
2002 Gender Identity, Sexual Orientation, and Sexual Diversity of DES Sons.
2001 A psycho-endocrinological overview of transsexualism.
2001 Environmental xenoestrogens, antiandrogens and disorders of male sexual differentiation.
1999 Intersexuality and gender identity differentiation.
1999 book : Sexual Differentiation of the Brain.
1999 The avian egg as a test system for endocrine disrupters: effects of diethylstilbestrol and ethynylestradiol on sex organ development.
1999 Transsexualism: a review of etiology, diagnosis and treatment.
1998 Effect of prenatal exposure to diethylstilbestrol on Müllerian duct development in fetal male mice.
1998 Sexual differentiation and environmental endocrine disrupters.
1996 Gender-identity, body-experience, sexuality, and the wish for having children in DES-daughters.
1996 Sexual differentiation of cognitive abilities in women exposed to diethylstilbestrol (DES) prenatally.
1996 The Effects of Diethylstilbestrol (DES) before Birth on the Development of Masculine Behavior in Juvenile Female Rhesus Monkeys.
1995 A quantitative investigation of gonadal feminization by diethylstilboestrol of genetically male embryos of the quail Coturnix coturnix japonica.
1995 A Sex Difference in the Human Brain and its Relation to Transsexuality.
1994 Chemically-Induced Alterations in Sexual and Functional Development: The Wildlife/Human Connection.
1994 Molecular feminization of mouse seminal vesicle by prenatal exposure to diethylstilbestrol: altered expression of messenger RNA.
1993 Gender-related behavior in women exposed prenatally to diethylstilbestrol.
1991 Gender-related behavior development in females exposed to diethylstilbestrol (DES) in utero: an attempted replication.
1991 Hormonal contributions to sexually dimorphic behavioral development in humans.
1990 Ingestion of diethylstilbestrol during pregnancy. Its responsibility in the testicular feminization syndrome.
1989 Female gene expression in the seminal vesicle of mice after prenatal exposure to diethylstilbestrol.
1989 The development of gender-related behavior in females following prenatal exposure to diethylstilbestrol (DES).
1988 Prenatal exposure of male mice to diethylstilbestrol alter the expression of the lactotransferrin gene in seminal vesicles.
1985 Antiandrogenic treatment of a gender-dysphoric transvestite.
1985 Effects of estrogen treatment on sexual behavior in male-to-female transsexuals: experimental and clinical observations.
1979 Feminization of the quail by early diethylstilbestrol treatment: histoenzymological investigations on steroid dehydrogenases in the gonads.
1978 Sex-dimorphic behaviour development in the human: prenatal hormone administration and postnatal socialization.
1977 Prenatal Exposure to Synthetic Progestins and Estrogens: Effects on Human Development.
1976 Pulmonary embolism in a transsexual man taking diethylstilbestrol.
1972 book : Man and Woman, Boy and Girl – the differentiation and dimorphism of gender identity from conception to maturity.
1959 Male Pseudohermaphrodism – Report of a Case, with Observations on Pathogenesis.
1959 Masculinization of the female infant associated with estrogenic therapy alone during gestation : four cases.



Edited 3 time(s). Last edit at 11/12/2019 06:01PM by Tai.

[www.hormonesmatter.com]

Maternal DES Exposure and Intersex Development in Males
Author: Hugh Easton

In my ongoing research into in connections between maternal DES exposure and male sexual development, I recently obtained a copy of the 1953 Physician’s Desk Reference (PDR), a compendium of pharmaceutical products listed together with their manufacturer’s recommended usage.

“Physician’s Desk Reference is published annually by Medical Economics, Inc., through the courtesy of the manufacturers whose major products are described in section 4. It is distributed to over 130,000 active practicing physicians and to the pharmacies and libraries of over 4,000 hospitals throughout the United States and its possessions.”

On page 464 is pharmaceutical giant Eli Lilly’s entry for DES, which I’ve scanned and inserted below. Several other manufacturers have their own branded version of DES (for instance, Boyle & Co’s “Hi-Bestrol” DES in 25mg uncoated tablets, for threatened and habitual abortion, supplied in bottles of 100 and 1,000). However, I will concentrate on Lilly’s entry, since they were the major manufacturer and distributor of the drug, and have provided a lot more detailed directions for prescribing the drug than the other manufacturers.
Uses and Dosing for DES According to its Manufacturer
DES Dosing 1953
Figure 1. DES dosing from 1953 Physician’s Desk Reference.

Lilly specify a variety of uses for DES: Menopause, “senile vaginitis”, painful engorgement of breasts, functional uterine bleeding, carcinoma of the prostate, and to prevent “accidents of pregnancy” (miscarriages). In addition, they helpfully provided a recommended dosing schedule, based on the one devised by Drs. George and Olive Smith, two of the chief proponents of the use of DES to prevent miscarriages. The dosing schedule involves a progressively increasing exposure to the drug, starting at 5mg per day, and progressively increasing as the pregnancy continues until it reaches 125mg per day in week 35 (I guess because, after week 35, the baby is sufficiently well developed that it doesn’t matter if it is born early).
High Dose DES and the Male Fetus

DES is one of the most powerful estrogens ever developed, and even 5mg represents a high dose. You can tell that by the way most of the other recommended uses involve doses considerably smaller. Treating the symptoms of menopause, for example, involves doses of 0.1 to 1 mg per day. Treatment of prostate cancer (through chemical castration) involves a starting dose of 3mg per day, and a maintenance dose (once testosterone suppression has been achieved) of 1mg per day.

Particularly during the later stages of prenatal development, a male fetus whose mother was given DES according to this schedule, was being exposed to many times the dose of DES required to cause complete suppression of testosterone production in an adult man. This is very important, because male development, and masculinization of the brain, are driven by the action of testosterone produced in a male fetus’s testicles. Prevent testosterone from being produced, and a genetically male fetus will develop as female instead of male, despite the Y chromosome. This was easily demonstrated by conditions such as Swyer’s syndrome and Complete Androgen Insensitivity Syndrome, in which a failure to produce testosterone or a failure to respond to the hormone, results in a person who is genetically male but physically female.

The conventional causes of intersex all tend to act throughout prenatal development. What I believe has happened with DES is that it has caused a form of intersex, but one that is different from any of the usual causes of intersex because it allowed relatively normal male development to take place during the first trimester, but from the end of the first trimester onward, DES exposure was high enough for testosterone production to become profoundly suppressed.

As you can see from the table, by the end of the first trimester, the dose had already reached 20mg per day and was still climbing. Presumably, the placenta provided some protection from the drug, or an early stage fetus is more resistant to chemical castration by DES than an adult man, because 20mg is already several times the induction dose for chemical castration of prostate cancer patients.

male genital development
Figure 2. Male reproductive development. Melmed, S., 2011. Williams textbook of endocrinology. Elsevier Health Sciences.

In the figure below, a chart from an endocrinology textbook showing a timeline of events associated with male physical development. Notice that male external genital differentiation begins in week 7 and has finished by the end of week 12.

During the time genital differentiation is taking place, the main things going on in the brain are: very rapid cell division, and migration of those cells to their final position in the brain (which is often far distant from where they formed).
fetal brain development
Figure 3. Fetal brain development. Andersen, S.L., 2003. Trajectories of brain development: point of vulnerability or window of opportunity?. Neuroscience & Biobehavioral Reviews, 27(1), pp.3-18.

The first elements of the permanent structure of the brain don’t start to be built until about 16 weeks after conception, by which time some brain cells have reached their final position and can start to form permanent connections with other brain cells (“Axonal/Dendritic outgrowth”). I’m guessing that there are two ways of wiring up brain tissue: a male way and a female way, and if there isn’t testosterone present during the time axonal and dendritic growth are taking place, you get the female version.

A process of programmed cell death starts soon after that, which may also be important for whether you end up with a male or female brain. Perhaps there are certain cells that generate aggressiveness and competitiveness, and they only stay alive when excess brain cells are being removed if there is testosterone present. I look at the way men’s faces light up when they’re watching competitive sports, and it’s pretty obvious that they’re experiencing something that I cannot. It makes me think that there could actually be cells in their brains that generate that “joy of sport”, which were culled from my brain because there wasn’t any testosterone present when their thumbs up or thumbs down moment arrived.

From these two diagrams, it is evident that the first trimester is the key time for genital development, while brain develop with regards to gender comes much later and corresponds with the ever increasing dosages of DES. With these progressively increasing doses, along with the known effects of synthetic hormones on brain development, a good argument can be made that DES may have producing people who are genetically male and look male, but have female brains. From talking to people with a known or suspected history of DES exposure, it certainly looks like that is what happens.

DES was used in somewhere in the region of 10 million pregnancies worldwide. The schedule published in the PDR wasn’t used in all of them by any means, but it was the manufacturer’s recommendation, and many doctors must surely have followed that recommendation. This raises the prospect that there could well be several million people alive today who look male but have female brains, as a result of the use of DES



Edited 1 time(s). Last edit at 11/12/2019 06:12PM by Tai.

[www.hormonesmatter.com]

November 7, 2019
Maternal DES and Male Sexual Development
Author: Hugh Easton

In the course of trying to figure out why I have certain symptoms normally associated with intersex conditions, but do not fit the criteria for any of the conventional genetic causes of intersex, I have stumbled across what appears to be a gigantic medical and pharmaceutical industry disaster that has been quietly unfolding ever since WWII. This post is my latest attempt to bring the problem to wider public attention.

DES: A Hormone for Every Ailment

In the years during and immediately after WWII, rapid advances in chemistry suddenly made it possible to produce man-made versions of many of the hormones that occur naturally in the human body. Hormones control a wide range of biological processes involved in the day to day running of the human body, and are also key drivers of the development and maintenance of masculine and feminine attributes.

Doctors and the pharmaceutical industry quickly realised that hormones had tremendous potential as medicines, and they were rushed to market with comparatively little safety testing, and even in spite of clear evidence of harm during what animal experiments were performed.

The earliest of these substances was an artificial estrogen called diethylstilbestrol (or DES). DES was developed by a team of chemists in the UK in 1938. It is a completely man-made substance that has a chemical structure unrelated to naturally occurring estrogens. Nonetheless, the DES molecule has a similar shape to natural estrogen molecules, binds really well to estrogen receptors and acts as an extremely potent estrogen (one of the most powerful ever developed). Furthermore, it is active when taken by mouth, and it is comparatively easy to synthesize, so can be manufactured in bulk very cheaply. DES had all the right characteristics to become a blockbuster drug.

Because it was developed using public funds, DES was never patented. The people who developed it hoped that, by making the secret of its manufacture freely available, it would make a low cost source of estrogen available to women who needed it. However, US pharmaceutical giant Eli Lilly spotted a moneymaking opportunity, and quickly made DES their own, becoming the main manufacturer and distributor of the drug worldwide throughout the time it was used as a medicine

With this new drug in their armory, Lilly needed to find uses for it. One of the first uses they came up with was in pregnancy, as a treatment for preventing miscarriages and premature births. Their theory was that one main cause of miscarriage was inadequate hormone production, and that by supplementing a woman’s own hormones with DES, she would be less likely to miscarry. Never mind that the animal research showed no reduction in miscarriages,

“Dodds was always against the automatic prescribing of estrogen for any reason. He was sickened when he first heard about the work of Dr Karl John Karnaky in Houston, the first to use DES widely to “prevent miscarriages”. Dodds sent Karnaky a study that he himself had performed, showing that in rabbits and rats, the drug caused miscarriages.” (p37 of Barbara Seaman’s “The Greatest Experiment”)

More importantly, there was a whole series of experiments carried out between 1938 and 1941 at Chicago’s Northwestern University showing that DES causes female development in male rat fetuses!
[www.ncbi.nlm.nih.gov]

Consequences of Maternal DES

Undeterred, Eli Lilly pressed ahead, and through an aggressive marketing campaign and with a bit of arm twisting at the FDA, soon had DES licensed as a wonder drug for preventing miscarriages. Between 1940 and about 1980 (by which time it had largely been withdrawn from use as a miscarriage preventative), DES was used in somewhere in the region of 10 million pregnancies worldwide . In the US, it is estimated that 4.8 million children were exposed prior to 1971 when the FDA withdrew its approval, although doctors continued to use it off-label for several years after (p14 “DES Voices”, Pat Cody).

“It is estimated that DES was prescribed to between 2 and 10 million pregnant women world-wide as pills, injections, suppositories, and creams to prevent miscarriage between 1947–1971 … The exact number of women/fetuses prenatally-exposed to DES world-wide is unknown (IARC 2012). The majority of reports of DES use are from the U.S. where it is believed that between the 1940s and 1970s, 5 to 10 million people either consumed DES during pregnancy or experienced in utero exposures (IARC 2012). DES use was also popular in Europe and Australia, and like the U.S., many women did not know that they were taking DES. Therefore, estimated numbers of people reporting exposure during pregnancy or in utero are around 300,000 in the United Kingdom and 200,000 in France (Harris and Waring 2012).”

DES Daughters

The “DES daughters” from those pregnancies have since been acknowledged to have suffered all kinds of problems as a result of their exposure, including: very high rates of several kinds of cancer; infertility; abnormalities of their internal reproductive organs; several times greater risk of miscarriage and ectopic pregnancies; and various other health problems, including autoimmune disorders, osteopenia, degenerative disc disease, endometriosis, premature menopause, the list goes on and on. Everyone I have talked to who was prenatally exposed to DES seems to have health problems of one kind or another attributable to their exposure.
DES Sons

In contrast with the daughters, the official line has always been that the “DES sons” suffered virtually no ill effects as a result of their exposure. Based on what I’ve seen since first finding out about DES in 2011, that is not true at all, and in fact what it has done to us is exactly the same thing it did to the rats at Chicago’s Northwestern University: it caused us to develop as female instead of male during the time it was being administered.

Under the standard dosing schedule, increasing doses of DES was given to women with doses of across pregnancy reaching a 125mg in the last month of pregnancy (Physician’s Desk Reference, 1953). As a result of this graduated hormone exposure, we might expect relatively normal male development during the first trimester, but predominantly female development during the second and third trimesters. The first trimester is when genital takes place and physical sexual attributes develop. During the second and third trimesters, the main things still ongoing as far as development is concerned, is brain development. With this graduated dose of synthetic estrogens, we would expect relatively normal genital development although there is emerging evidence of alterations here too, but feminized brain development. In other words, with DES sons, we see a person who looks male but whose brain has predominantly or overwhelmingly, in a fair number of cases it seems, developed as female. The way this tends to manifest itself later in life is as a person who everyone regards as male, but who has a strong inner sense of being a woman.

Other common effects associated with DES exposure in males include subfertility or infertility, and hypogonadism (chronic below normal male testosterone production). I suspect this may also be a big risk factor for testicular cancer, however, this is unproven. All the genuine research into DES effects on males appears to have been discontinued by about 1980, and the total number of people studied is too small to assess cancer risk.
In Utero Synthetic Progestins

DES isn’t the only man-made hormone with gender bending properties to have seen widespread use during pregnancy. There is a class of hormones called progestins (which are all man-made versions of the hormone progesterone), that have also seen extensive use for miscarriage prevention. The first progestins were actually derivatives of testosterone. Although they act more like progesterone in adult women, in female fetuses they turned out to act more like testosterone. Female babies who had partially developed as male were being born throughout the 1950s as a result of progestin exposure. I have found a number of papers all published around 1960 reporting on cases of “progestin induced virilization”, so that appears to be when the medical community first realized there was a problem with these drugs. How long these androgenic progestins continued beyond this point I don’t know, nor do I know how many pregnancies they were used in in total. However, in her book “DES: The Complete Story”, the author, Cynthia Laitman, describes how she was given an androgenizing progestin alongside DES during her pregnancy in 1969, so it appears these gender bending drugs were being used throughout the 1960s too.

Unlike DES, progestins were never withdrawn. They are the main ingredient in birth control pills and other forms of hormonal contraception, and taken by several hundred million women every day for contraception. In fact some progestin formulations are still used for miscarriage prevention (allegedly these are non-androgenizing types of progestin, however, I think there’s a big risk they could be inducing female brain development in male babies).

In the course of trying to find out about the effects of DES on the unborn child, I came across a group of people whose babies were harmed by another hormone-based medicine, a drug called Primodos (marketed as Duogynon in Germany). A similar drug, called Gestest, was marketed in the United States. Here, the exposure occurred very early in the pregnancy, during the time organogenesis and limb development is taking place in the fetus, and before the process of sexual development is underway. As a result, the main effects have been severe, thalidomide like disabilities rather than abnormalities of sexual development (although, at least one member of their Facebook group had a Primodos exposed brother who was trans identified).

As with DES and the androgenizing progestins debacles, the pharmaceutical industry has been given a free pass by the governments of the countries involved, and the whole thing swept under the rug. An important fact about Primodos is that it contains exactly the same hormones as are used in birth control pills, the only difference being that the dose is higher.

A third group of people I’ve come across whose lives have been blighted by prenatal exposure to synthetic hormones, are in Hhorages France . The main issue this group is trying to highlight is that many of their hormone exposed children later committed suicide or developed serious psychiatric illnesses (although reading some of their published research, it appears that many of the children have intersex related abnormalities too).

My hope is that by alerting people to the harm so many have suffered as a result of being exposed in the womb to medically prescribed hormones, I can help to prevent such exposures from taking place in the future. I am also hoping this post and my Facebook page, Protect the unborn child from synthetic hormones, will help gain greater public acceptance for the transgender community; a much maligned group of people who, through no fault of their own, were born with brains that do not properly match their biological sex.



Edited 1 time(s). Last edit at 11/12/2019 06:42PM by Tai.

[www.hormonesmatter.com]

By Hugh Easton

I found a pair of slides illustrating an important scientific discovery made in 1959, which became known as the Organizational-Activational hypothesis. A team of scientists experimenting on guinea pigs, discovered that hormones early in life play a crucial role in determining the sex of the brain, and whether an animal will have male or female behavior in adult life. Similar results have since been found in numerous other animal species, and there is little doubt that the same applies to human beings too. Actually there is no doubt at all, going by my own experiences.

Basically the brain has a sex, which arises during a critical period in prenatal development (or in rats, which are born at an earlier stage of development than humans, during days 1-10 of postnatal life). If there are high levels of androgenic hormones present during this critical time, the brain becomes masculinized, and the adult behavior will be that of a male. If there aren’t androgenic hormones present, the brain develops following the default female pathway instead, and the adult behavior will be that of a female.

Rats are a particularly convenient animal for demonstrating this because their critical period occurs after birth. This makes it relatively straightforward to produce a male rat with a female brain through surgical castration at birth. With most other animals you’d have to do the surgery in utero, which is a much trickier task. It is also relatively straightforward to determine whether a particular rat has a male or female brain, because those with male brains when injected with testosterone will attempt to mate with any females present, whereas those with female brains will not. In addition, rats with female brains will adopt a posture called the lordosis posture when injected with estradiol, whereas those with male brains will not.
Organizational and Activational Effects of Hormones during Pregnancy

organizational and activational effects - males

organizational and activational effects

In the first graphic shown above, we see what happens to the adult behavior of males put through three different treatments:

castrated as an adult
castrated at birth with no supplemental testosterone
castrated at birth, but with supplemental testosterone administered.

As is illustrated, treatments 1 and 3 result in adults that behave as males, whereas treatment 2 results in adults with female behavior. What this shows is that there is a crucial period early in the postnatal life of a rat during which either the presence of functioning testicles or externally administered testosterone causes male brain development to occur. In the absence of either, the rat ends up with a female brain, and female adult behavior despite being physically and genetically male. The period of development is roughly equivalent to gestational week 20 through birth during human pregnancy.

The second graphic illustrates what happens to females similarly treated. In this case through spaying, or removal of the ovaries, rather than castration. Here, only treatment 3 (spaying at birth + administering supplemental testosterone) results in an adult with male behavior. This is because, ovaries don’t produce much testosterone. So even the females spayed as adults do not have much testosterone present during the critical period for the sex of their brain. Once again, this emphasizes that it is the presence of testosterone during a critical period in brain development that produces a male brain, and the absence of that hormone during the critical period in which the brain ends up female instead. It appears that genetics and the sex of the body have nothing to do with sex based brain development. Instead, the sex of the brain is entirely dependent on whether or not androgenic hormones were present during a critical period in its development.

There are a couple of other things we can take away from this. Firstly, the effects of hormones during that critical period are permanent, and thus, are present for the remainder of the animal’s life. Secondly, anything that results in males being castrated, or females being exposed to testosterone-like hormones, during that critical period is a really bad thing, and likely to result in a male with a female brain or female with a male brain.
DES and Other Hormones Administered During Pregnancy

The miscarriage prevention drug DES given to women from the 1940s through the 1970s is a powerful chemical castration agent, so it is no small wonder that so many of the male-assigned people from DES pregnancies show signs of female brain development. The first generation progestins ethisterone and norethisterone, widely used in the 1950s and 1960s for miscarriage prevention, are both testosterone derivatives that have been shown to masculinize female fetuses (Wilkins 1960). This is why I think there’s likely to be a significant population of female assigned people from that era with male brains, as a result of their use.

DES isn’t the only drug that acts as a chemical castration agent to have been used during pregnancy. Progestins can also act as chemical castration agents, and unlike DES, they were never withdrawn, but remain in use even now. The archetypal progestin, medroxyprogesterone acetate (MPA), is one of the most popular drugs for chemical castration of sex offenders. It is in my 1972 Monthly Index of Medical Specialties (MIMS) and 1981 British National Formulary for treatment of habitual abortion (i.e. miscarriage prevention), so has definitely been used in human pregnancies in the past.

More to the point, hydroxyprogesterone caproate, which seems to be the most popular currently used miscarriage preventative, has similar pharmacological properties to MPA. The primary difference is that it is a pure progestin that doesn’t cross-react with non-target receptor types, whereas MPA, to a small extent, does. Other than that, they are quite similar drugs with similar effects. Therefore, I think there is a big risk of male assigned children from pregnancies in which hydroxyprogesterone caproate is used, being born with female brains. It is certainly something that bears looking at, by people independent of the pharmaceutical industry and the medical establishment



Edited 1 time(s). Last edit at 11/12/2019 06:49PM by Tai.

My Painkillers Turned Me Gay

[www.youtube.com]

This man proved to himself that a painkiller called pregabalin turned him gay. He started to have same sex attraction about 4-5 days on the medication. When he stopped the painkiller, his attraction to his girlfriend returned.

Pregabalin side effects may include
[www.drugs.com]
Endocrine & metabolic: Fluid retention (2% to 3%), hypoglycemia (2% to 3%), decreased libido (?1%)

Genitourinary: Urinary incontinence (1% to 3%), impotence (?1%), urinary frequency (?1%), erectile dysfunction (?1%), urinary tract infection (1%)

<1%, postmarketing, and/or case reports
breast hypertrophy, ejaculatory disorder, gynecomastia,heavy menstrual bleeding
hirsutism, increased libido, lactation (females)

• Suicidal ideation: Pooled analysis of trials involving various antiepileptics (regardless of indication) showed an increased risk of suicidal thoughts/behavior (incidence rate: 0.43% treated patients compared to 0.24% of patients receiving placebo); risk observed as early as 1 week after initiation and continued through duration of trials (most trials ?24 weeks). Monitor all patients for notable changes in behavior that might indicate suicidal thoughts or depression; notify healthcare provider immediately if symptoms occur.



Edited 1 time(s). Last edit at 11/17/2019 05:56AM by Tai.

A study done in the midwest, which is where atrazine is in the drinking water (see above...atrazine makes some male frogs into females) shows some straight men having gay thoughts with enough alcohol.

[pubs.niaaa.nih.gov]

Alcoholic Beverages as a Source of Estrogens
Judith S. Gavaler, Ph.D.
Alcoholic beverages contain not only alcohol but also numerous other substances (i.e., con-geners) that may contribute to the beverages’ physiological effects. Plants used to produce alcoholic beverages contain estrogen-like substances (i.e., phytoestrogens). Observations that men with alcoholic cirrhosis often show testicular failure and symptoms of feminization have suggested that alcoholic beverages may contain biologically active phytoestrogens as con-geners. Biochemical analyses have identified several phytoestrogens in the congeners of bourbon, beer, and wine. Studies using subjects who produced no estrogen themselves (i.e.,rats whose ovaries had been removed and postmenopausal women) demonstrated that phyto-estrogens in alcoholic beverage congeners exerted estrogenlike effects in both animals and humans. Those effects were observed even at moderate drinking levels.

[www.tandfonline.com]

Sexual willingness with same- and other-sex prospective partners: Experimental evidence from the bar scene
Breanne R. Helmers, Colin R. Harbke & Julie C. Herbstrith ORCID Icon
Pages 109-124 |

ABSTRACT

Sex is ubiquitous in the media, but only a fraction depicts sexual interactions between same-sex partners. This field study, conducted outside of bars in the Midwestern United States, examined 83 heterosexuals’ sexual willingness with a same- or other-sex partner. Participants viewed a randomly assigned video vignette of a same- or other-sex partner. Alcohol intake, partner attractiveness, and sexual willingness were measured. Using moderated regression analysis, we found that alcohol intake predicted sexual willingness with the male target for both men and women, but not with the female target. The attractiveness of same-sex partners was related to sexual willingness. Sexual willingness was only influenced by alcohol intake and perceived attractiveness of a same-sex prospective partner. Most notably, alcohol intake was related to increased sexual willingness of men with a same-sex partner, suggesting a potential shift in normative casual sexual behavior among heterosexual men.

Here are a couple of snippets from my file on Sex...

[vimeo.com]
AGENDA: Grinding America Down
1:31:39 Minute Video

AGENDA Grinding America Down (Full Movie)
1:31:39 Minute Video
[www.youtube.com]

JR’s Notes:

…

11:02 MM
“The book was The Naked Communist by Cleon Skousen who had been a former FBI Agent and inside the book it documented 45 Current Communist Goals from 1958.” Curtis Bowers 11:16 MM

12:05 MM
Goal #26:
Present Homo-Sexuality, Degeneracy, and Promiscuity as “Normal, Natural and Healthy.”

38:44 MM
The Homo-Sexual Movement

“I’m a student of Communism and the Communist set up various Groups and Societies. Their Society that they set up to Promote Homosexuality in this Country was called the Mattachine Society. It was founded by Henry Hay, a leading member of the Communist Party. So since I was studying Communism and teaching on the issue of Communism, you just follow leads and all of a sudden you realize what is this Mattachine? I never heard of this Mattachine Society. Well, it was Henry Hay’s organization set up to infiltrate the Culture of the United States to make Homo-Sexuality Normal.” Dr. David Noebel 39:21 MM

…
---------------------------------------------------------------------------------
[www.henrymakow.com]
The Mass Media as Human Pesticide
October 17, 2009
by Henry Makow Ph.D.

In the documentary, "Demographic Bomb," the Director of the UN Population Division, Hania Zlotnick admits the UN got women in developing countries to sterilize themselves by having TV stars do it in soap operas.

In developed countries, the Illuminati bankers have achieved the same goal in similar fashion by pushing promiscuity and homosexuality in the mass media.

In a famous 1969 memo, Rockefeller-sponsored Planned Parenthood VP Frederick Jaffe proposed the following measures to reduce U.S. fertility: (a) encourage increased homosexuality,(b) fertility control agents in water supply, (c) encourage women to work, (d) abortion and sterilization on demand, and (e) make contraception truly available and accessible to all.
[famous 1969 memo - [www.illuminati-news.com] - see below]

The mass media is the mental "water supply." It defines "norms" (acceptable behavior) and therefore reshapes behavior. You can't watch television without seeing aggressive career women showing up men and engaging in promiscuous sex, often with each other. Homosexuality is treated as normal, the "cool" and "edgy" thing. The Illuminati have moved the goalposts, and created a new bogus reality. They have decreased the birth rate in the United States as surely as in Bangladesh.

An illustration of how tightly controlled the TV plot-lines are: This week, 90 television shows are promoting "volunteerism" in their stories. They don't tell you what else they have promoting over the years, but it's plain to see: racism, i.e. the disappearance of race through interracial marriages; disappearance of religion and family (feminism); and the aforementioned promiscuity and homosexuality. This agenda is mirrored in the school system.

…
---------------------------------------------------------------------------------
[www.illuminati-news.com]
Dialectics, Rockefellers, and Population Control (Part 2)
by Dr. Dennis L. Cuddy, NewsWithViews, Nov 27, 2006

…

17) March 11, 1969---Vice-President of Planned Parenthood-World Population Frederick Jaffe's "Activities Relevant to the Study of Population Policy for the U.S." is printed containing a memo to Population Council president Bernard Berelson. It includes examples of proposed measures to reduce U.S. fertility, such as (a) encourage increased homosexuality, (b) fertility control agents in water supply, (c) encourage women to work, (d) abortion and sterilization on demand, and (e) make contraception truly available and accessible to all.

…



Edited 1 time(s). Last edit at 11/19/2019 01:44PM by John Rose.