[nutritionfacts.org]

Quote

The association between fat and insulin resistance is now widely accepted, so-called ectopic fat accumulation, the accumulation of fat in places it’s not supposed to be within our muscle cells. But not all fats affect the muscles the same The type of fat, saturated or unsaturated, is critical. Saturated fats like palmitate, found mostly in meat, dairy and eggs, cause insulin resistance, but oleate, found mostly in nuts, olives, and avocados may actually improve insulin sensitivity. What makes saturated fat bad? Saturated fat causes more of those toxic breakdown products and mitochondrial dysfunction and increase oxidative stress free radicals and inflammation, establishing a vicious cycle of events in which saturated fat induced free radicals causes dysfunction in the little powerplants within our muscle cells, which causes an increase in free production and impairment of insulin signaling.

Fat cells filled with saturated fat activate an inflammatory response to a far greater extent. This increased inflammation, along with eating more saturated fat, has been demonstrated to raise insulin resistance through free radical and ceramide production. Saturated fat also has been shown to have a direct effect on skeletal muscle insulin resistance. Accumulation of saturated far increases the amount of diacyl- glycerol in the muscles, which has been demonstrated to have a potent effect on muscle insulin resistance. It doesn’t matter if the fat in our blood comes from our fat, or their fat.

You can take muscle biopsies from people and correlate the saturated fat buildup in their muscles with insulin resistance.

While monounsaturated fats are more likely to be detoxified or safely stored away, saturated fats create those toxic breakdown products like ceramide that causes lipotoxicity. Lipo meaning fat, as in liposuction, and toxicity. This fat toxicity in our muscles is a well-known concept in the explanation of trigger for insulin resistance.

I’ve talked about the role saturated and trans fats contribute to the progression of other diseases, like autoimmune diseases, cancer and heart disease, but they can also cause insulin resistance, the underlying cause of prediabetes and type 2 diabetes. In the human diet, saturated fats are derived from animal sources while trans fats originate in meat and milk, in addition to partially hydrogenated and refined vegetable oils.

That’s why experimentally shifting people from animal fats to plant fats can improve insulin sensitivity. Insulin sensitivity was impaired on the diet with added butterfat but not on the diet with added olive fat.

We know prolonged exposure of our muscles to high levels of fat leads to severe insulin resistance, with saturated fats demonstrated to be the worst, but they don’t just lead to inhibition of insulin signaling, the activation of inflammatory pathways, and the increase in free radicals, they cause an alteration in gene expression, leading to a suppression of key mitochondrial enzymes like carnitine palmitoyltransferase, which finally solves the mystery of why those eating vegetarian have a 60% higher expression of that fat burning enzyme. They’re eating less saturated fat.

So do those eating plant-based diets have less fat clogging their muscles and less insulin resistance too? There hasn’t been any data available regarding the insulin sensitivity or inside muscle cell fat of those eating vegan or vegetarian, until now.

Researchers at the Imperial College of London compared the insulin resistance and muscle fat of vegans versus omnivores. Now those eating plant-based diets have the unfair advantage of being so much slimmer, so they found omnivores who were as skinny as vegans to see if plant-based diets had a direct benefit, as opposed to indirectly pulling fat out of the muscles by helping people lose weight in general.

They found significantly less fat trapped in the muscle cells of vegans compared to omnivores at the same body weight, better insulin sensitivity, better blood sugar levels, better insulin levels and, excitingly, significantly improved beta-cell function, the cells in the pancreas that make the insulin. They conclude that eating vegan is not only expected to be cardioprotective, helping prevent our #1 killer, heart disease, but that veganism is beta-cell protective as well, helping also to prevent our seventh leading cause of death, diabetes.

Funny, I didn't read anything about extra-virgin coconut oil in that entire article - looks a lot like someone intentionally wrote a deceptive forum subject. Not all saturated fats are equal but I definitely don't expect that to stop you from creating more threads on coconut/coconut oil in the future under the false impression that you are posting accurate data.

I read this quickly...maybe I missed it. Does it even mention the difference betwee fat treated with heat and normal fat, natural one, not treated with anything? Or does the 'study' not really care if we heat the fat to million degrees or ate it natural?

Huh?

yawn...
(please give graham some coconut oil)

Yes, coconut fat, high fat plants and plant oils do cause insulin resistance, but we can make the higher fat diet safe so this does not occur. I recently wrote about this on my site and posted studies on how we can greatly reduce insulin resistance and inflammation when eating high fat.

I'll give you a hint....build up the long chain omega 3 fatty acids in the body to greatly increase insulin sensitivity so we can better deal with those fats. Broccoli sprouts will also go a long way in helping.

Once again, Dr Gregor is not telling us the full story, none of the low fat promotors ever seen to tell us the truth....always partial truth but never the whole truth. Dr Gregor posts lots of studies, but he doesn't always seem to be able to put all the information togeather properly to be able to use it for strategic purposes....he has lots of holes in his ideas.

www.thesproutarian.com



Edited 2 time(s). Last edit at 02/09/2015 11:30PM by The Sproutarian Man.

I thought it was common knowledge that coconut oil is saturated fat. But for the usual deniers,

[www.cadiresearch.org]

Quote

Coconut oil has the unique distinction of having the highest proportion of saturated fat (92%) and cholesterol-raising (12-16 carbons) SAFA (74%) compared to 53% in butter fat and 24-29% in the beef, pork, lamb, and chicken fat.2, 4-6 Specifically, coconut oil contains 47% lauric (C12), 18% myristic (C14), and 9% palmitic (C16) acid.2, 4-6.

Coconut is the principal agent used for producing experimental atherosclerosis.10 Waqar et al. have demonstrated that the intake of saturated fat in the form of coconut oil with a normal number of calories is detrimental to glucose and lipid metabolism.10 The enhanced atherosclerosis on rabbits fed 26% energy from coconut compared to 15% and normal chow is given in Figure 069. 10

Once again, not all saturated fat is the same.

They are medium-chain fatty acids and are raw, unlike cooked animal products.


[articles.mercola.com]

Even though coconut oil is high in saturated fat, populations consuming large quantities of coconut products (such as in the Philippines), have low rates of heart disease—but this is likely due to their overall diet—with a very low consumption of meats (cholesterol) and processed foods and high intakes of rice and vegetables—rather than the coconut.

[www.drmcdougall.com]

i am not taking a position here, just posting it.

[www.ncbi.nlm.nih.gov] - "The results demonstrated the potential beneficiary effect of virgin coconut oil in lowering lipid levels in serum and tissues and LDL oxidation by physiological oxidants. This property of VCO may be attributed to the biologically active polyphenol components present in the oil."

[www.ncbi.nlm.nih.gov] - "Consumption of a diet rich in MCTs results in greater loss of AT compared with LCTs, perhaps due to increased energy expenditure and fat oxidation observed with MCT intake. Thus, MCTs may be considered as agents that aid in the prevention of obesity or potentially stimulate weight loss."

[www.ncbi.nlm.nih.gov] - "Arteriosclerosis is thought to be part of the normal aging process which is due to age associated molecular biological changes. The lipid theory of arteriosclerosis is rejected. Pertinent studies with MCT include these observations. Feeding of MCT to rats resulted in animals of low body weight, small fat deposits and excellent survival rate. This deserves emphasis because of the beneficial influence of low body weight on aging and arteriosclerosis. MCT feeding was associated with low linoleate and low tocopherol requirements in rats. This may lead to reduced formation of those linoleate derived prostaglandins which favor thrombosis formation. Lower linoleate requirements may also lead to the presence of fewer uncontrolled free radicals in the cells. MCT feeding is associated with low levels of serum and liver cholesterol involving speculations that tissue conditions are such that an adaptive increase of cholesterol is unnecessary. The Demographic Yearbook of the United Nations (1978) reported that Sri Lanka has the lowest death rate from ischemic heart disease. Sri Lanka is the only of the countries giving reliable data where coconut oil (containing over 50% medium chain fatty acids) is the main dietary fat."

[www.ncbi.nlm.nih.gov] - "These findings suggest that medium-chain triglyceride application to spontaneously hypertensive rats improves decreased cardiac function and cardiac hypertrophy without affecting blood pressure and myocardial mRNA expression of molecular markers."

Panchito Wrote:
-------------------------------------------------------
> I thought it was common knowledge that coconut oil
> is saturated fat. But for the usual deniers,
>
> [www.cadiresearch.org]
> -coconut
>
> Coconut oil has the unique distinction of having
> the highest proportion of saturated fat (92%) and
> cholesterol-raising (12-16 carbons) SAFA (74%)
> compared to 53% in butter fat and 24-29% in the
> beef, pork, lamb, and chicken fat.2, 4-6
> Specifically, coconut oil contains 47% lauric
> (C12), 18% myristic (C14), and 9% palmitic (C16)
> acid.2, 4-6.
>
> Coconut is the principal agent used for producing
> experimental atherosclerosis.10 Waqar et al. have
> demonstrated that the intake of saturated fat in
> the form of coconut oil with a normal number of
> calories is detrimental to glucose and lipid
> metabolism.10 The enhanced atherosclerosis on
> rabbits fed 26% energy from coconut compared to
> 15% and normal chow is given in Figure 069. 10
>


The point l was trying to make was that saturated fat (animal or plant) does not cause insulin resistance when one has good levels of EPA/DHA in the diet. If we have good levels of EPA/DHA in the body we should be able to handle the higher fat diets.

I had a big meal of chia sprouts with extra coconut fat today (2 tablespoons). After four hours my blood sugar is 4.8, so it is far from being high. In contrast, when l have fruit it can go high.

In coming days l will do an experiement and have 2 tablespoons of coconut fat with omega 6 sunflower. If my expectations are correct my blood sugar should still be stable because l would have good reserves of the long chain omega 3s.

Makes me wonder how Robert Cassar and Lou Carona go. They don't seem to do the high omega 3 foods.

www.thesproutarian.com



Edited 2 time(s). Last edit at 02/10/2015 07:25AM by The Sproutarian Man.

I do know that Cassar does massive amounts of coconut oil...

You should stop posting this, it's not showing what you think it shows.

jtprindl Wrote:
-------------------------------------------------------
> [www.ncbi.nlm.nih.gov] - "The
> results demonstrated the potential beneficiary
> effect of virgin coconut oil in lowering lipid
> levels in serum and tissues and LDL oxidation by
> physiological oxidants. This property of VCO may
> be attributed to the biologically active
> polyphenol components present in the oil."

This one found only that raw was not as bad as cooked.


> [www.ncbi.nlm.nih.gov] -
> "Consumption of a diet rich in MCTs results in
> greater loss of AT compared with LCTs, perhaps due
> to increased energy expenditure and fat oxidation
> observed with MCT intake. Thus, MCTs may be
> considered as agents that aid in the prevention of
> obesity or potentially stimulate weight loss."


Fat % loss on MCT: 0.67%
Fat % loss on olive oil: 0.2%

The difference is about 1/2 of a percent. Visually and
health-wise, this is an imperceptible difference.


> [www.ncbi.nlm.nih.gov] -
> "Arteriosclerosis is thought to be part of the
> normal aging process which is due to age
> associated molecular biological changes. The lipid
> theory of arteriosclerosis is rejected. Pertinent
> studies with MCT include these observations.
> Feeding of MCT to rats resulted in animals of low
> body weight, small fat deposits and excellent
> survival rate. This deserves emphasis because of
> the beneficial influence of low body weight on
> aging and arteriosclerosis. MCT feeding was
> associated with low linoleate and low tocopherol
> requirements in rats. This may lead to reduced
> formation of those linoleate derived
> prostaglandins which favor thrombosis formation.
> Lower linoleate requirements may also lead to the
> presence of fewer uncontrolled free radicals in
> the cells. MCT feeding is associated with low
> levels of serum and liver cholesterol involving
> speculations that tissue conditions are such that
> an adaptive increase of cholesterol is
> unnecessary. The Demographic Yearbook of the
> United Nations (1978) reported that Sri Lanka has
> the lowest death rate from ischemic heart disease.
> Sri Lanka is the only of the countries giving
> reliable data where coconut oil (containing over
> 50% medium chain fatty acids) is the main dietary
> fat."


I have other data about the Sri Lankans:

The recent estimates for mortality from cardio and cerebrovascular diseases (CVD) for Sri Lanka--524 deaths per 100,000--is higher than that observed in many Western economies. (ref: Atherosclerosis. 2003 Dec;171(2):157-61)

You are not doing yourself any favors by eating a lot of this stuff.

But if you are intent on making yourself sicker and fatter than you have to be, keep on eating a lot of it.

Cassar is lean and mean, not an ounce of fat on his frame. His skin is translucent, smooth and tight. Explain that.

"This one found only that raw was not as bad as cooked."

Looks a lot more like it found the conclusion I posted - ""The results demonstrated the potential beneficiary effect of virgin coconut oil in lowering lipid levels in serum and tissues and LDL oxidation by physiological oxidants. This property of VCO may be attributed to the biologically active polyphenol components present in the oil". I don't expect you to admit it, though, as you have a clear bias towards oils and fat.


[www.ncbi.nlm.nih.gov] - "In conclusion, VCO is a cheap oil source containing high concentration of MCFAs which in the current study had shown beneficial effect in WC reduction especially in males without any deleterious effects to the lipid profile. VCO is also safe to use for the period of study without any deleterious effects on biochemical and organ functions."

[www.ncbi.nlm.nih.gov] - "This study showed that VCO supplementation is capable of preventing elevation in blood pressure and also decreasing deactivation of nitric oxide in male rats fed with repeatedly heated palm oil. In addition, VCO does not influence relaxation but decreases vasoconstriction of the endothelium".

Dietary intake of saturated fat by food source and incident cardiovascular disease: the Multi-Ethnic Study of Atherosclerosis - "Although dietary recommendations have focused on restricting saturated fat (SF) consumption to reduce cardiovascular disease (CVD) risk, evidence from prospective studies has not supported a strong link between total SF intake and CVD events... A higher intake of dairy SF was associated with LOWER CVD risk. In contrast, a higher intake of meat SF was associated with GREATER CVD risk. The substitution of 2% of energy from meat SF with energy from dairy SF was associated with a 25% lower CVD risk. No associations were observed plant or butter SF and CVD risk". - [www.ncbi.nlm.nih.gov]

Cholesterol, coconuts, and diet on Polynesian atolls: a natural experiment: the Pukapuka and Tokelau island studies - "Two populations of Polynesians living on atolls near the equator provide an opportunity to investigate the relative effects of saturated fat and dietary cholesterol in determining serum cholesterol levels. The habitual diets of the toll dwellers from both Pukapuka and Tokelau are high in saturated fat but low in dietary cholesterol and sucrose. Coconut is the chief source of energy for both groups. Tokelauans obtain a much higher percentage of energy from coconut than the Pukapukans, 63% compared with 34%, so their intake of saturated fat is higher. The serum cholesterol levels are 35 to 40 mg higher in Tokelauans than in Pukapukans. These major differences in serum cholesterol levels are considered to be due to the higher saturated fat intake of the Tokelauans. Analysis of a variety of food samples, and human fat biopsies show a high lauric (12:0) and myristic (14:0) content. Vascular disease is uncommon in both populations and there is no evidence of the high saturated fat intake having a harmful effect in these populations". - [www.ncbi.nlm.nih.gov]

Coconut fats - "Coconut fats account for 80% of the fat intake among Sri Lankans. Around 92% of these fats are saturated fats. This has lead to the belief that coconut fats are 'bad for health', particularly in relation to ischaemic heart disease. Yet most of the saturated fats in coconut are medium chain fatty acids whose properties and metabolism are different to those of animal origin. Medium chain fatty acids do not undergo degradation and re-esterification processes and are directly used in the body to produce energy. They are not as 'bad for health' as saturated fats. There is the need to clarify issues relating to intake of coconut fats and health, more particularly for populations that still depend on coconut fats for much of their fat intake. This paper describes the metabolism of coconut fats and its potential benefits, and attempts to highlight its benefits to remove certain misconceptions regarding its use". - [www.ncbi.nlm.nih.gov]

Dietary intake and the risk of coronary heart disease among the coconut-consuming Minangkabau in West Sumatra, Indonesia - "Similar intakes of saturated and unsaturated fatty acids between the cases and controls indicated that the consumption of total fat or saturated fat, including that from coconut, was not a predictor for CHD in this food culture. However, the intakes of animal foods, total protein, dietary cholesterol and less plant derived carbohydrates were predictors of CHD". - [www.ncbi.nlm.nih.gov]

[www.ncbi.nlm.nih.gov] - "The results imply no specific role for coconut or coconut oil in the causation of CHD in the present set of Indian patients from Kerala."

>I don't expect you to admit it, though, as you have a clear bias towards oils and fat.

perhaps you mean a bias "against" oils and fat?

and maybe i could rewrite the sentence for you
"I don't expect myself to admit it, though, since I have a clear bias against ever admitting that I don't know anything or have ever made an error of any kind, ever"
jtprindl

NuNativs Wrote:
-------------------------------------------------------
> Cassar is lean and mean, not an ounce of fat on
> his frame. His skin is translucent, smooth and
> tight. Explain that.

He is a fasting instructor. People that eat a high fat diet hide this important information. They need fasting to compensate. Some call it intermittent like Mercola. Not only that. They also eat very low calories as compared to normal people which makes then skinny. That is the explanation also for the Atkins diet.

[www.hawaiimedicalfasting.com]

NuNativs Wrote:
-------------------------------------------------------
> Cassar is lean and mean, not an ounce of fat on
> his frame. His skin is translucent, smooth and
> tight. Explain that.

he eats milk and eggs and salts, superfoods, oils, etc

he looks good. explaining it would be due to low caloric intake while eating fats. no big deal. fat intake must be taken into account with caloric intake.


inteesting video

[www.youtube.com]



also.........
i cannot verify the truth of this - posted on discussion group

That's Dr. Robert Cassar, a former MD who injects himself daily with expensive HGH and claims we can live on superfood powders. Most people who go the superfoods route of powders and stimulants end up worse than SAD eaters. He only recommends these powders because he's been paid by a company to promote their products. He definitely has a impressive physique (see video below) but what you see is the result of steroids and hgh not superfoods. But people see his body and automatically associate it with the products he's promoting.

fresh Wrote:
-------------------------------------------------------

>
> perhaps you mean a bias "against" oils and fat?
>
> and maybe i could rewrite the sentence for you
> "I don't expect myself to admit it, though, since
> I have a clear bias against ever admitting that I
> don't know anything or have ever made an error of
> any kind, ever"


Heh heh, thanks Fresh.

Nobody ever provided convincing evidence that oils of
any kind are a health food.

If one wants to eat a concentrated source of fat, the
logical choices are nuts, seeds, avocado, olive, and
maybe even a bit of coconut now and then. Or durian,
even, which has a lot more fat than most fruits.

But oils, no. No way, no how, never. I use them
myself (EVOO is my choice), but I do not kid myself
that I am doing myself any favors.

They are just relatively empty calories. But even among
empty calorie sources, there are better and worse choices.
Considering about 60 years of epidemiology and clinical
trials, the preponderance of evidence still stands: saturated
fats should be limited. Even if they are raw. Nothing magic
happens because they are raw. They just aren't as damaged
as cooked.

Claims that you must follow a low-fat diet are nothing more than simple-minded, scientifically unsupported, dogmatic beliefs. There is zero evidence that raw, plant-based fats are unhealthy in any way, shape, or form. In fact, there's lots of evidence to the contrary. Many people claim that fat in general is atherogenic, completely oblivious to the fact that there's a huge difference between raw, plant-based fats and cooked animal fats. It is also a myth that saturated fat causes heart disease. Inflammation causes heart disease - fats cooked at high temperatures and excessive intake of omega-6's are both highly inflammatory. Fat is not the bad guy, inflammation is. Omega-3's are powerful anti-inflammatory fats which offer significant protection against atherosclerosis and heart disease and help promote a healthy, balanced omega fatty acid ratio. Furthermore, there's an overwhelming amount of scientific evidence proving the heart healthy benefits of fatty foods such as nuts, seeds, and extra-virgin olive oil. For anyone under the assumption that there is credible evidence suggesting that fat in general is to blame for heart disease, it's very important to consider the aforementioned inflammatory factors as well as the quality of food used in the study. For example, conflicting results have been reported when comparing extra-virgin olive oil to regular olive oil, with extra-virgin olive oil demonstrating a myriad of health benefits. Obviously, highly refined and/or highly processed foods aren't going to give favorable results. This fat condemning logic is on par with condemning all vegan diets because of a study that found poor results with vegans eating processed/junk food diets but decided to mindlessly place all the blame on veganism.

Cassar does eat eggs but milk no. I have never heard about him taking steroids, he's pretty nazi about food, it doesn't seem something he would do, but you never no...

"They need fasting to compensate."

This is nothing other than an opinion based on zero credible evidence.

fresh Wrote:
-------------------------------------------------------
> Even though coconut oil is high in saturated fat,
> populations consuming large quantities of coconut
> products (such as in the Philippines), have low
> rates of heart disease—but this is likely due to
> their overall diet—with a very low consumption
> of meats (cholesterol) and processed foods and
> high intakes of rice and vegetables—rather than
> the coconut.
>
> [www.drmcdougall.com]
> t.htm
>
> i am not taking a position here, just posting it.


Or, maybe, just maybe, it is because they eat a lot of raw shells, crustaceans and fish? Maybe?

jtprindl Wrote:
-------------------------------------------------------
> Claims that you must follow a low-fat diet are
> nothing more than simple-minded, scientifically
> unsupported, dogmatic beliefs.

oh, prindl, so strident, so intense!

do you think it possible for you to post just once without using the words simple or dogmatic?
or would you need to use some other language other than english?

for what it is worth, although others may have an opinion on the matter, I do not maintain that raw plant based fats are unhealthy.

what i would ask though is for you to do an experiment.

eat 100% raw oil for a month and provide us with health indicators from a doctor before and after.

because i could easily do the same for all veggie diet, or all fruit diet with no worries.

yes i know it's not an exact equivalent. no matter. would be fun.


or perhaps suez could submit to a battery of health tests for our pleasure.





There is zero
> evidence that raw, plant-based fats are unhealthy
> in any way, shape, or form. In fact, there's lots
> of evidence to the contrary. Many people claim
> that fat in general is atherogenic, completely
> oblivious to the fact that there's a huge
> difference between raw, plant-based fats and
> cooked animal fats. It is also a myth that
> saturated fat causes heart disease. Inflammation
> causes heart disease - fats cooked at high
> temperatures and excessive intake of omega-6's are
> both highly inflammatory. Fat is not the bad guy,
> inflammation is. Omega-3's are powerful
> anti-inflammatory fats which offer significant
> protection against atherosclerosis and heart
> disease and help promote a healthy, balanced omega
> fatty acid ratio. Furthermore, there's an
> overwhelming amount of scientific evidence proving
> the heart healthy benefits of fatty foods such as
> nuts, seeds, and extra-virgin olive oil. For
> anyone under the assumption that there is credible
> evidence suggesting that fat in general is to
> blame for heart disease, it's very important to
> consider the aforementioned inflammatory factors
> as well as the quality of food used in the study.
> For example, conflicting results have been
> reported when comparing extra-virgin olive oil to
> regular olive oil, with extra-virgin olive oil
> demonstrating a myriad of health benefits.
> Obviously, highly refined and/or highly processed
> foods aren't going to give favorable results. This
> fat condemning logic is on par with condemning all
> vegan diets because of a study that found poor
> results with vegans eating processed/junk food
> diets but decided to mindlessly place all the
> blame on veganism.

jtprindl Wrote:
-------------------------------------------------------
> "They need fasting to compensate."
>
> This is nothing other than an opinion based on
> zero credible evidence.

The scientific search for the secret of The Atkins Diet BBC Documentary (49:03):

[www.youtube.com]

Shortcut: Eat all you want. But then you find you don't want to eat because of the effects of food on appetite. You undereat (low calory) and lose weight.

Mercola: every day new claims on the benefits of fasting. Eat fat. Fasting this and that. Some people on high fat may only eat once a day.

Eating fat doesn't make you fat so fasting in order to not gain weight on high-fat diets is not necessary at all. And after all this time, you still haven't figured out that 1.) not all high-fat diets are the same (such as raw diets versus the Atkins diet) and 2.) high-fat does not equal ketogenic.

Panchito wrote:
"Some people on high fat may only eat once a day."

And then you have the contrary with Durianrider stuffing his face with huge volumes of food all his waking hours or he withers away...

Maybe there's a happy medium...

There's also nothing wrong with only eating once per day if you are getting all the nutrition you need (and preferably lots of phytochemicals). In fact, it would be beneficial because your digestive system wouldn't have to work nearly as hard.

within less than two hours after a meal, the body goes into fasting mode. If a person eats only once a day on a high fat diet, then they fast for ~21 hrs of the day or 87.5% of the day. Fasting has drawbacks and benefits. It is not all gold. It could lead to muscle wasting.

[en.wikipedia.org]

Quote

Starvation mode is a state in which the body is responding to prolonged periods of low energy intake levels. During short periods of energy abstinence, the human body will burn primarily free fatty acids from body fat stores. After prolonged periods of starvation the body has depleted its body fat and begins to burn lean tissue and muscle as a fuel source.

Ordinarily, the body responds to reduced energy intake by burning fat reserves and consuming muscle and other tissues. Specifically, the body burns fat after first exhausting the contents of the digestive tract along with glycogen reserves stored in muscle and liver cells.[2] After prolonged periods of starvation, the body will utilize the proteins within muscle tissue as a fuel source. People who practice fasting on a regular basis, such as those adhering to energy restricted diets, can prime their bodies to abstain from food while reducing the amount of muscle burned.[3]

"It could lead to muscle wasting."

Muscle wasting is not a concern with intermittent fasting.


"within less than two hours after a meal, the body goes into fasting mode."

Not true at all - many foods aren't even completely digested within two hours.

Dr. Esselstyn's famous shouting "NO OIL" routine. He's intimidating.

[www.youtube.com]